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Authors(2023, Int J Mol Sci)
Year2023
JournalInternational Journal of Molecular Sciences
Typeprimary
Tieremerging
Ingested2026-05-08
View published source (10.3390/ijms24033000) →

2023 — HERV-W ENV → linc01930/cGAS axis → neuronal apoptosis

One-paragraph summary

Direct experimental confirmation of the HERV-W ENV ↔ cGAS interaction predicted by the project's HERV-Mitochondrial-Inflammation Loop hypothesis. Demonstrates: (a) cGAS interacts with HERV-W ENV; (b) HERV-W ENV triggers IFN-β expression; (c) HERV-W ENV → cGAS pathway drives neuronal apoptosis in recent-onset schizophrenia patients; (d) the long non-coding RNA linc01930 mediates the HERV-W ENV → cGAS axis. Validates Q40 pipeline mechanism hypothesis at the protein-protein interaction level — provides experimental ground truth for the pipeline's Step 6 docking. Cross-condition support: same HERV-W ENV → cGAS axis operating in schizophrenia subset suggests it's a general post-viral neuroinflammation mechanism rather than disease-specific.

Claims as triples

Methods note

Cell-line and patient-sample studies. Recent-onset schizophrenia patient PBMCs/serum. Gene-expression analysis identifying linc01930 as mediator. Western blot, IP, functional assays for HERV-W ENV ↔ cGAS interaction.

Limitations

Triangulation notes

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